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33 1 2011 1 JournalofNingxiaMedicalUniversity 27 :1674-6309(2011)01-0027-05 = *+,-./ 01c,, F, GH, IJ (9, 750004) 1 :, - `? A,? >.@A, + 1275`? - ` 48.6%; 50.3%, 40.8% W (P<0.01) ` A,TC W TG;(P<0.01) W TC.W TG U\]#VW. TC TGU\]#\],HDLC W W WHR9 BMI D? 6. 2 - ` W, # [ # N <` 345: `; ;; 6789:R446.11 :;:A `0,. 4 D % DE 12h@ AG # [1] U- O. W9 2,=> 9 @.P.\], `. =< + 5> \]. 1.3 `5O - `,N.: K( TC 5.72mmol L -1,\$ < % 9! ", 2008 5 M TG 1.70mmol L -1, W`( K( 2008 11 A%H HDL-C 0.91mmol L -1, <1.01mmol L -1, 1 # ` ` 2C : 1.1 % A.,A 2C,X@2 7.0mmol L -1 ;W 9, 25* _CJ 5. J - 93 : W,X(SBP) 140mmHg 9(X)(DBP) 90mmHg; # : (BMI) 24kg/m 2, H (WC). 1.2 85cm, 80cm [2] ; 1.2.1? 1H: - (WHR): 0.95 0.8 SY N " ;20 BYB 2 CZ[YB 1 :W I 92C _;,5*,5 3_(5 ;3 O@ :YB, 5!5 "59 5) 5,E EO 1.4 [ ZC,%) 1.2.2 = W # 0,. W # >,? $ #?`$ 1.2.3 > 2 ` a D 1.5 + " >"; =0,W :2010-03-15 <= :+*(NZ0771) />?@: (1962-),,,W<,' < [41 AB/>:,NO,,' < [ 41 _ =1;: =0, =1;YB =1, =0;,5 =1, =0;BMI9 WC>";W =1, =0;2C =1, =0; 2C =1, =0;W =1, =0 1.6 ;@A " SPSS11.5 ;,; χ 2 =<, G.

28 33 t=<, G. @ 2.2 `! Logistic` W TC W TG HDLC. 2 10.6% 45.5% 7.6%,W TC 2.1 H 1564, 1275 W TG HDLC., 851, 424,(41.48± 14.2% 19.8% 5.0% A,χ 2 " 11.68)?` 619, 8.417 80.02 3.244,TC W TG 48.6%; 50.3%, 40.8% A;(P<0.01) χ 2 = ` W,A < Y, W TC.W TG (χ 2 =10.23,P<0.001),; U\]#VW., 1 1 &2 01, / n TC TG HDL /% /% /% 25~ 229 14 6.1 89 38.9 17 7.4 35~ 405 42 10.4 210 51.9 33 8.1 45~ 134 23 17.2 60 44.8 9 6.7 55~ 55 9 16.4 20 36.4 4 7.3 60_ 28 2 7.1 8 28.6 2 7.1 ; 851 90 10.6 387 45.5 65 7.6 χ 2 " 12.318 0.730 0.041 P" 0.000 0.393 0.840 25~ 85 2 2.4 6 7.1 5 5.9 35~ 112 9 8.0 20 17.9 8 7.1 45~ 95 20 21.1 25 26.3 1 1.1 55~ 101 25 24.8 38 37.6 7 6.9 60_ 31 13 41.9 18 58.1 0 0.0 ; 424 69 14.2 84 19.8 21 5.0 χ 2 " 32.629 54.164 2.960 P" 0.000 0.000 0.085 P<0.01 2.3 `,TG 55 _U \]#\],TC 35 ~G W ;,TC TG U\]#\];55 TC TG;HDLC W, 2 2.4 Logistic` Logistic`:;. ` YB, 5 BMI WHR 2C W ;#`./0, 3 2.5 Logistic`.,%;. 9, Logistic`, ;. ;!`A78,! 9%.=< а=0.05, ; 4 `78,3 : W WHR9 BMI? 6.,?/0, 4

1, - `? 29 2 &2 03,( x±s,mmol L 珋 -1 ) / n TC TG HDLC 25~ 229 1.851±1.624 4.120±1.163 1.241±0.371 35~ 405 2.221±1.806 4.530±1.010 1.311±0.476 45~ 134 2.187±1.563 4.875±0.987 1.282±0.390 55~ 55 1.971±1.261 4.674±0.983 1.268±0.398 60_ 28 1.811±1.623 4.830±1.070 1.312±0.343 F" 4.068 13.131 1.231 P" 0.003 0.000 0.298 25~ 85 1.114±0.523 4.099±0.817 1.387±0.413 35~ 112 1.659±1.517 4.562±1.016 1.326±0.493 45~ 95 1.748±1.008 4.889±0.990 1.436±0.340 55~ 101 2.069±1.354 5.010±1.063 1.417±0.659 60_ 31 2.039±0.902 5.391±0.969 1.431±0.324 F" 10.317 19.322 0.093 P" 0.000 0.000 0.985 3 LogisticL 8 B BE(B) Waldχ 2 P" OR(95%CI) ` 0.371 0.118 9.943 0.002 1.450(1.151~1.826) 0.096 0.133 0.518 0.472 1.101(0.848~1.430) -0.429 0.110 15.238 0.000 0.651(0.525~0.808) YB 0.256 0.093 7.528 0.006 0.774(0.645~0.930),5 0.263 0.100 6.937 0.008 0.769(0.632~0.935) BMI 1.148 0.108 112.107 0.000 3.151(2.548~3.897),& -0.131 0.103 1.638 0.201 0.877(0.717~1.072) -0.256 0.104 6.043 0.014 0.774(0.631~0.949) W 0.195 0.133 2.166 0.141 0.823(0.634~1.067) 2C 0.115 0.092 1.562 0.211 0.891(0.744~1.067) WHR 1.109 0.167 44.289 0.000 3.032(2.187~4.204) 2C 0.959 0.306 9.809 0.002 0.383(0.210~0.698) W 0.802 0.185 18.706 0.000 0.449(0.312~0.645) 4 LogisticL 8 B SE(B) Waldχ 2 P" OR(95%CI) -0.386 0.119 10.436 0.001 0.68(0.538~0.859) W 0.612 0.200 9.346 0.002 0.542(0.366~0.803) WHR 0.707 0.176 16.136 0.000 2.028(1.436~2.863) BMI 1.025 0.113 81.615 0.000 2.787(2.231~3.481) 3 I2 3Y: - ` 48.6%, 50.3%, 40.8%,W 18.6% 22.% 15.9% [3], W ' 46.50% [4],Q 59.3% [5] ` W,A;,D.,aC ). N9.E,C+ /,Q?!;W TC W TG HDLC. 10 6% 45.5% 7.6%,W TC W TG HDLC. 14.2% 19 8% 5.0% W TC 55_W,W TG9 HDLC, 4 [6] ) χ 2 =< Y,W TC W TG. U

30 33 #\]. `.. ` 8,,TG 54 _U \]#\],TC 35~G W ;,TC TG U\] #\];55 TC TG ;HDLCW, D ; 6U [7],_., ^GN W, & `. ;# Z,- `. YB,5 BMI WHR 2C W ;#`./0,?.< YB,59 # `. 91'. O @^1.3. O,YB,5!`.# " [8] YB,5. ` Y W YB 3, YB. - 42.7%,,5. - 40.5%, O1\] `., 9PQ O@ N. <@A`. ^ 6 [9-10] # `., + ` W :*, H`a`,3? 3 * $\],^E.b,, SN D,T\] [11],! ` - ] (B 31.2%, - EO 9 (,? 45 G Logistic` W WHR9 BMI? 6. ` )-.#, 1? 41#, [9. < * 56 O@,,E +) ]$ ( [ # W, [, U` 0 '. JK :: [1] ` 2[,1 - N. ` 2 [J]. H, 2007,35(5):390. [2]? 41G " 1G. #9%0.<> D" #9.[J]. H,2002,23(1):5-10. [3] H,.,$ /,. 18 _ ` [J]. H<,2005,39 (5):306-310. [4] 0H, 5,,.' ;1- `? [J].<, 2008,15(2):327-331. [5], 8,. 35-74. ` [J].<, 2004,5(2):118-120. [6] 4,,H,.- ` [J].0 [,2006,10(5): 485-488. [7] 8,%, ',.; 15- ` [J].H2<,2006, 32(5):1-5. [8] B5.2- YB,5 W `. [J]. H,2003,12:1115-1117. [9] BrownCD,H igginsm,donatoka,etal.bodymas indexandtheprevalenceofhypertensionanddyslipidem ia[j].obesres,2000,8(9):605. [10] F,,) 8,. #% ` 29. [J].0 3 >,2003,18 (9):348. [11] 456,B, 27,.W # / 8.[J]. H,2002,23(6):466-469. (FG:H)

1, - `? 31 PrevalenceandRiskFactorsofDyslipidemia amongadultsinyinchuan LIUXiu-ying,LIULan,ZHAOYi,ZHANGYu-hong,HUJi-hong (Dept.ofNutrition,NingxiaMed.Univ.,Yinchuan,750004) Abstract:Objective Toexploretheprevalenceandinfluencingfactorsoflipidabnormalitiesamongadultin habitantsinyinchuanandtoprovidethetheorybasisforcommunityinterventionofcardiovasculardiseases. Methods Serumlipidsandphysicalindicesof1275urbanadultsweresurveyedinyinchuan.Dyslipidemia prevalenceandtherelatedriskfactorswereanalyzed.results Theprevalenceofdyslipidemiawas48.6% in adultresidentsofyinchuanandmaleandfemaleprevalencewere50.3% and40.8% respectivly.tcandtg inmalesweresignificanthigherthanthoseinfemales(p<0.01).thedyslipidemiaprevalenceincreased withagerisinginbothofmalesandfemales.hypertension,bodymasindexandthewhrposiblywerethe independentriskfactorsofdyslipidemia.conclusion Dyslipidemiaprevalenceofadultsinyinchuanwere higherthanthatofnationwide.themiddle-agedandelderlypeoplearekeycontrolgroups.controlingboth weightandbloodpresurelevelsunderthenormalrangecanbeefectiveforpreventingdyslipidemia. Keywords:dyslipidemia;prevalence;riskfactors;Yinchuan ("# 26 ) [7] G,9::. caga oipa icea1. =>? [J]. = <,2004,14 (2):133-134. [8] MarcDB,MathewAS,RubenG,etal.Focaladhe sionkinaseproteinlevelsingutepithelialmotility[j]. AmJPhysiolGastrointestLiverPhysiol,2006,291(9): G491-G499. [9] SuJM,GuiL,ZhouYP,etal.Expresionoffocaladhe sionkinaseandalpha5andbetalintegrinsincarcinomas anditsclinicalsignifieanee[j].worldjgastroenterol, 2002,8(4):613-618. (FG:H) TheRelationshipbetweenOipAandtheExpresionofFAK ingastriccancertisue JIChun-wei 1,CHENGDong-xian 2,YANGLi 3 (1.NingxiaMed.Univ.,Yinchuan750004; 2.HospitalofTraditionalChineseMedicineinShizuishan City,Shizuishan753000; 3.TheAfiliatedHospitalofNingxiaMed.Univ.,Yinchuan750004) Abstract:Objective ToexploretherelationshipbetweenoipAandtheexpresionofFAKinchronicgastric diseaseandgastriccacerinfectedanditssignificance.methods Gastricbiopsieswerecolectedfromchronic atrophicgastritis(cag)andgastriccancer(gc).theexpresionoffakweredetectedbyimmunohisto- chemistry.oipagenewasdetctedbypcr.results TheaveragedetectionrateofoipA genewas66.3% (59/89);theratewas56.5%(26/46)amongCAG,and76%(33/43)amongGC.TheexpresionsofFAKin HPoipA+groupweresignificantlyhigherthanthoseinHPoipA-group(P<0.05)inpatientswithCAGand GC;TheexpresionsofFAKinGCweresignificantlyhigherthanthoseinCAG.Conclusion OipAgeneof Helicobacterpylori(HP)isrelatedwiththeexpresionsofFAKinchronicgastricdiseaseandgastriccancer. Keywords:focaladhesionkinase;helicobacterpylori;outerimflammatoryproteingenotype;chronicgastric disease;gastriccancer

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